
Obstructive sleep apnoea (OSA) occurs when the upper airway repeatedly narrows or closes during sleep, causing reduced airflow or breathing pauses. Sleep becomes fragmented and blood oxygen may fall. Not everyone with OSA experiences pronounced daytime sleepiness.
Snoring alone does not establish OSA, and some people with OSA do not snore loudly.
Obesity, increasing age, family history and certain jaw or pharyngeal anatomical features raise the likelihood of OSA. Large tonsils may contribute. A deviated nasal septum can impair nasal breathing, but is not usually the sole cause of OSA.
OSA is associated with increased risk of hypertension, cardiovascular disease, metabolic problems and concentration difficulties. Excessive sleepiness can increase the risk of accidents, particularly when driving. Individual risk depends on severity, other medical conditions and additional factors.
Assessment combines a clinical history, examination of the upper airway and an appropriate sleep study, either a home sleep-apnoea test in selected uncomplicated adults or in-laboratory polysomnography. Testing establishes severity and guides treatment. An ENT examination can identify anatomical contributors but does not replace a sleep study.
Septoplasty can improve nasal breathing or CPAP tolerance, but is not, on its own, a reliable cure for obstructive sleep apnoea.
Persistent snoring with breathing pauses, excessive sleepiness or unrefreshing sleep warrants assessment by a sleep-medicine clinician, often alongside ENT evaluation.